Neonates with respiratory distress syndrome (RDS), and children and adults with acute respiratory distress syndrome (ARDS) often require supplemental oxygen and mechanical ventilation because of decreased arterial PO 2, which by itself may induce further ventilator induced lung injury (VILI) and result in significant mortality and morbidity. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.ĭata Availability: All relevant data are within the paper and its Supporting Information file.įunding: Work performed in this study was supported in part by the Department of Defense/Office of Naval Research N000141210597 and N000141210810 (MRW).Ĭompeting interests: The authors have declared that no competing interests exist. Received: JAccepted: JanuPublished: February 3, 2016Ĭopyright: © 2016 Hummler et al. PLoS ONE 11(2):Įditor: Edgardo Szyld, Icahn School of Medicine at Mount Sinai, ARGENTINA ![]() (2016) The Effects of Lung Protective Ventilation or Hypercapnic Acidosis on Gas Exchange and Lung Injury in Surfactant Deficient Rabbits. ![]() ![]() Citation: Hummler HD, Banke K, Wolfson MR, Buonocore G, Ebsen M, Bernhard W, et al.
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